β-Adrenoceptor modulation of transiently overexpressed α2-adrenoceptors in brain and peripheral tissues: cellular mechanisms underlying the developmental toxicity of terbutaline-Reference-Cited by-同舟云学术

β-Adrenoceptor modulation of transiently overexpressed α2-adrenoceptors in brain and peripheral tissues: cellular mechanisms underlying the developmental toxicity of terbutaline

Published:2004-01 Issue:4 Volume:62 Page:305-314
ISSN:0361-9230
Container-title:Brain Research Bulletin
language:en
Short-container-title:Brain Research Bulletin

Author:

Kreider Marisa L,Seidler Frederic J,Slotkin Theodore A

Publisher

Elsevier BV

Subject

General Neuroscience

Reference72 articles.

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3. β-Adrenoceptor-mediated cell signaling in the neonatal heart and liver: responses to terbutaline;Auman;Am. J. Physiol.,2001

4. Are developing β-adrenoceptors able to desensitize? Acute and chronic effects of β-agonists in neonatal heart and liver;Auman;Am. J. Physiol.,2002

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3. Cross-talk from β-Adrenergic Receptors Modulates α2A-Adrenergic Receptor Endocytosis in Sympathetic Neurons via Protein Kinase A and Spinophilin;Journal of Biological Chemistry;2013-10

4. Terbutaline impairs the development of peripheral noradrenergic projections: Potential implications for autism spectrum disorders and pharmacotherapy of preterm labor;Neurotoxicology and Teratology;2013-03

5. Physiological and pathophysiological roles of placental aminopeptidase in maternal sera: possible relation to preeclampsia and preterm delivery;Expert Opinion on Medical Diagnostics;2009-07-29