High-energy compounds mobilize intracellular Ca2+ and activate calpain in cultured cells: Is calpain an energy-dependent protease?-Reference-Cited by-同舟云学术

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High-energy compounds mobilize intracellular Ca2+ and activate calpain in cultured cells: Is calpain an energy-dependent protease?

Published:2014-03 Issue: Volume:102 Page:9-14
ISSN:0361-9230
Container-title:Brain Research Bulletin
language:en
Short-container-title:Brain Research Bulletin

Author:

Nguyen Huey T.,Chen Ming

Publisher

Elsevier BV

Subject

General Neuroscience

Reference32 articles.

1. Creatine and its potential therapeutic value for targeting cellular energy impairment in neurodegenerative diseases;Adhihetty;Neuromol. Med.,2008

2. Calcium – a life and death signal;Berridge;Nature,1998

3. Nicotine induces calcium spikes in single nerve terminal varicosities: a role for intracellular calcium stores;Brain;Neuroscience,2001

4. The fateful encounter of mitochondria with calcium: how did it happen?;Carafoli;Biochim. Biophys. Acta,2010

5. Calpain inhibition: a therapeutic strategy targeting multiple disease states;Carragher;Curr. Pharm. Des.,2006

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. N -methyl- d -aspartate receptor-mediated calcium overload and endoplasmic reticulum stress are involved in interleukin-1beta-induced neuronal apoptosis in rat hippocampus;Journal of Neuroimmunology;2017-06

2. Our â€œenergy-Ca2+ signaling deficitsâ€ hypothesis and its explanatory potential for key features of Alzheimerâ€™s disease;Frontiers in Aging Neuroscience;2014-12-03

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