The α2δ-1-NMDAR1 interaction in the trigeminal ganglion contributes to orofacial ectopic pain following inferior alveolar nerve injury
Author:
Funder
National Natural Science Foundation of China
Department of Science and Technology of Sichuan Province
Publisher
Elsevier BV
Subject
General Neuroscience
Reference83 articles.
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1. Phosphorylation of the AMPARs regulated by protein kinase C (PKC) and protein interacting with C-kinase 1 (PICK1) contribute to orofacial neuropathic pain;Brain Research;2023-12
2. The α2δ-1-NMDA receptor complex and its potential as a therapeutic target for ischemic stroke;Frontiers in Neurology;2023-04-20
3. Metabotropic glutamate receptor 5‐mediated inhibition of inward‐rectifying K+ channel 4.1 contributes to orofacial ectopic mechanical allodynia following inferior alveolar nerve transection in male mice;Journal of Neuroscience Research;2023-02-18
4. Differential roles of NMDAR subunits 2A and 2B in mediating peripheral and central sensitization contributing to orofacial neuropathic pain;Brain, Behavior, and Immunity;2022-11
5. NMDARs mediate peripheral and central sensitization contributing to chronic orofacial pain;Frontiers in Cellular Neuroscience;2022-09-27
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