Brain–blood barrier breakdown and pro-inflammatory mediators in neonate rats submitted meningitis by Streptococcus pneumoniae

Author:

Barichello Tatiana,Fagundes Glauco D.,Generoso Jaqueline S.,Paula Moreira Ana,Costa Caroline S.,Zanatta Jessiele R.,Simões Lutiana R.,Petronilho Fabricia,Dal-Pizzol Felipe,Carvalho Vilela Márcia,Lucio Teixeira Antonio

Funder

CNPq, FAPESC, FAPEMIG, UNESC, INCT-TM

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference41 articles.

1. Antibiotic therapy prevents, in part, the oxidative stress in the rat brain after meningitis induced by Streptococcus pneumoniae;Barichello;Neurosci. Lett.,2010

2. Correlation between behavioral deficits and decreased brain-derived neurotrophic [correction of neurotrofic] factor in neonatal meningitis;Barichello;J. Neuroimmunol.,2010

3. TNF-alpha, IL-1beta, IL-6, and cinc-1 levels in rat brain after meningitis induced by Streptococcus pneumoniae;Barichello;J. Neuroimmunol.,2010

4. Pathophysiology of acute meningitis caused by Streptococcus pneumoniae and adjunctive therapy approaches;Barichello;Arq. Neuro-Psiquiatr.,2012

5. Circulating concentrations, cerebral output of the CINC-1 and blood–brain barrier disruption in Wistar rats after pneumococcal meningitis induction;Barichello;Eur. J. Clin. Microbiol. Infect. Dis.,2012

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