Sumo-2/3-ylation following in vitro modeled ischemia is reduced in delayed ischemic tolerance-Reference-Cited by-同舟云学术

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Sumo-2/3-ylation following in vitro modeled ischemia is reduced in delayed ischemic tolerance

Published:2009-05 Issue: Volume:1272 Page:71-80
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Loftus Liam T.,Gala Rosaria,Yang Tao,Jessick Veronica J.,Ashley Michelle D.,Ordonez Andrea N.,Thompson Simon J.,Simon Roger P.,Meller Robert

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference43 articles.

1. Ischemic preconditioning and brain tolerance: temporal histological and functional outcomes, protein synthesis requirement, and interleukin-1 receptor antagonist and early gene expression;Barone;Stroke,1998

2. A M55V polymorphism in a novel SUMO gene (SUMO-4) differentially activates heat shock transcription factors and is associated with susceptibility to type I diabetes mellitus;Bohren;J. Biol. Chem.,2004

3. Regulation of SUMOylation by reversible oxidation of SUMO conjugating enzymes;Bossis;Mol. Cell.,2006

4. Mammalian hibernation: cellular and molecular responses to depressed metabolism and low temperature;Carey;Physiol. Rev.,2003

5. Increased protein SUMOylation following focal cerebral ischemia;Cimarosti;Neuropharmacology,2008

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