Mild cognitive deficits associated to neocortical microgyria in mice with genetic deletion of cellular prion protein-Reference-Cited by-同舟云学术

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Mild cognitive deficits associated to neocortical microgyria in mice with genetic deletion of cellular prion protein

Published:2008-11 Issue: Volume:1241 Page:148-156
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Xikota João C.,Rial Daniel,Ruthes Daniel,Pereira Rafael,Figueiredo Cláudia P.,Prediger Rui D.S.,Walz Roger

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference62 articles.

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2. A synaptic model of memory: long-term potentiation in the hippocampus;Bliss;Nature,1993

3. Electrophysiological characteristics of reactive astrocytes in experimental cortical dysplasia;Bordey;J. Neurophysiol.,2001

4. Normal development and behaviour of mice lacking the neuronal cell-surface PrP protein;Bueler;Nature,1992

5. Cortical malformations and epilepsy: new insights from animal models;Chevassus-au-Louis;Epilepsia,1999

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1. Deficiency of prion protein induces impaired autophagic flux in neurons;Frontiers in Aging Neuroscience;2014-08-25

2. Deletion of the prion gene Prnp affects offensive aggression in mice;Behavioural Brain Research;2014-06

3. Unraveling the neuroprotective mechanisms of PrPCin excitotoxicity;Prion;2012-07

4. Overexpression of cellular prion protein (PrPC) prevents cognitive dysfunction and apoptotic neuronal cell death induced by amyloid-β (Aβ1–40) administration in mice;Neuroscience;2012-07

5. Cellular Prion and its Catabolites in the Brain: Production and Function;Current Molecular Medicine;2012-03-01

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