Copper deficiency results in AMP-activated protein kinase activation and acetylCoA carboxylase phosphorylation in rat cerebellum
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience
Reference41 articles.
1. Nitric oxide switches on glycolysis through the AMP protein kinase and 6-phosphofructo-2-kinase pathway;Almeida;Nat. Cell. Biol.,2004
2. Myocardial ischemia differentially regulates LKB1 and an alternate 5¢-AMP-activated protein kinase kinase;Altarejos;J. Biol. Chem.,2005
3. Abnormal development and increased 3-nitrotyrosine in copper-deficient mouse embryos;Beckers-Trapp;Free Radic. Biol. Med.,2006
4. Copper deficiency induces the upregulation of the copper chaperone for Cu/Zn superoxide dismutase in weanling male rats;Bertinato;J. Nutr.,2003
5. The AMP-activated protein kinase is involved in the regulation of ketone body production by astrocytes;Blazquez;J. Neurochem.,1999
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