Central substance P NK1 receptors are involved in fever induced by LPS but not by IL-1β and CCL3/MIP-1α in rats

Author:

Reis R.C.,Brito H.O.,Fraga D.,Cabrini D.A.,Zampronio A.R.

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

Fundação Araucária do Estado do Paraná

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference74 articles.

1. The effect of CP-96, 345, a non-peptide substance-P antagonist, on thermoregulation and the development of endotoxin-fever in rats;Balasko;J. Therm. Biol,2000

2. Human interleukin (IL) 1 alpha, murine IL-1 alpha and murine IL-1 beta are transported from blood to brain in the mouse by a shared saturable mechanism;Banks;J. Pharmacol. Exp. Ther.,1991

3. Penetration of interleukin-6 across the murine blood–brain barrier;Banks;Neurosci. Lett.,1994

4. Blockade of lipopolysaccharide-induced fever by a mu-opioid receptor-selective antagonist in rats;Benamar;Eur. J. Pharmacol.,2000

5. Substance P primes murine peritoneal macrophages for an augmented proinflammatory cytokine response to lipopolysaccharide;Berman;Neuroimmunomodulation,1996

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