Dose-dependent inhibition of GCPII to prevent and treat cognitive impairment in the EAE model of multiple sclerosis-Reference-Cited by-同舟云学术

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Dose-dependent inhibition of GCPII to prevent and treat cognitive impairment in the EAE model of multiple sclerosis

Published:2016-03 Issue: Volume:1635 Page:105-112
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Hollinger Kristen R.,Alt Jesse,Riehm Alison M.,Slusher Barbara S.,Kaplin Adam I.

Funder

National Multiple Sclerosis Society (AIK)

NIH

Bruce and Deborah Downey and Mulligans Fore MS

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference30 articles.

1. Proton MR spectroscopy-detectable major neurotransmitters of the brain: biology and possible clinical applications;Agarwal;AJNR Am. J. Neuroradiol.,2012

2. Relevance of cognitive deterioration in early relapsing-remitting MS: a 3-year follow-up study;Amato;Mult. Scler.,2010

3. Treatment of cognitive impairment in multiple sclerosis: position paper;Amato;J. Neurol.,2013

4. Identification of the N-glycosylation sites on glutamate carboxypeptidase II necessary for proteolytic activity;Barinka;Protein Sci.,2004

5. Lower brain glutamate is associated with cognitive deficits in HIV patients: a new mechanism for HIV-associated neurocognitive disorder;Ernst;J. Magn. Reson. Imaging.,2010

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