Isoflurane neurotoxicity involves activation of hypoxia inducible factor-1α via intracellular calcium in neonatal rodents-Reference-Cited by-同舟云学术

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Isoflurane neurotoxicity involves activation of hypoxia inducible factor-1α via intracellular calcium in neonatal rodents

Published:2016-12 Issue: Volume:1653 Page:39-50
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Chai Dongdong,Jiang Hong,Li Qifang

Funder

National Nature Science Foundation of China

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference50 articles.

1. Prosurvival and prodeath effects of hypoxia-inducible factor-1alpha stabilization in a murine hippocampal cell line;Aminova;J. Biol. Chem.,2005

2. Chelation of cellular calcium modulates hypoxia-inducible gene expression through activation of hypoxia-inducible factor-1alpha;Berchner-Pfannschmidt;J. Biol. Chem.,2004

3. The versatility and universality of calcium signalling;Berridge;Nat. Rev. Mol. Cell Biol.,2000

4. Briner A., De Roo, M., Dayer A., Muller D., Habre W., Vutskits L., 2010. Volatile anesthetics rapidly increase dendritic spine density in the rat medial prefrontal cortex during synaptogenesis. Anesthesiology 112, 546–556.

5. Cycling hypoxia induces chemoresistance through the activation of reactive oxygen species-mediated B-cell lymphoma extra-long pathway in glioblastoma multiforme;Chen;J. Transl. Med.,2015

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