Altered glutamate release in the dorsal striatum of the MitoPark mouse model of Parkinson's disease-Reference-Cited by-同舟云学术

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Altered glutamate release in the dorsal striatum of the MitoPark mouse model of Parkinson's disease

Published:2016-11 Issue: Volume:1651 Page:88-94
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Farrand Ariana Q.,Gregory Rebecca A.,Bäckman Cristina M.,Helke Kristi L.,Boger Heather A.

Funder

MUSC Barmore Fund

National Institutes of Health

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference41 articles.

1. Repeated amphetamine treatment causes a persistent elevation of glial fibrillary acidic protein in the caudate-putamen;Armstrong;Eur. J. Pharm.,2004

2. An update on the potential role of excitotoxicity in the pathogenesis of Parkinson’s disease;Blandini;Funct. Neurol.,2010

3. A partial GDNF depletion leads to earlier age-related deterioration of motor function and tyrosine hydroxylase expression in the substantia nigra;Boger;Exp. Neurol.,2006

4. Improved ceramic-based multisite microelectrode for rapid measurements of l-glutamate in the CNS;Burmeister;J. Neurosci. Methods,2002

5. Characterization of a mouse strain expressing Cre recombinase from the 3′ untranslated region of the dopamine transporter locus;Bäckman;Genesis,2006

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