Nicotinamide mononucleotide protects against β-amyloid oligomer-induced cognitive impairment and neuronal death-Reference-Cited by-同舟云学术

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Nicotinamide mononucleotide protects against β-amyloid oligomer-induced cognitive impairment and neuronal death

Published:2016-07 Issue: Volume:1643 Page:1-9
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Wang Xiaonan,Hu Xuejun,Yang Yang,Takata Toshihiro,Sakurai Takashi

Funder

Ministry of Education, Culture, Sports, Science, and Technology of Japan

Chinese National Foundation of National Sciences

Specialized Research Fund for the Doctoral Programme of Higher Education

Project of Science and Technology Department of Liaoning, China

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference43 articles.

1. Poly(ADP-ribose) polymerase-1-mediated cell death in astrocytes requires NAD+ depletion and mitochondrial permeability transition;Alano;J. Biol. Chem.,2004

2. NAD+ depletion is necessary and sufficient for poly(ADP-ribose) polymerase-1-mediated neuronal death;Alano;J. Neurosci.,2010

3. NAD+ metabolism in health and disease;Belenky;Trends Biochem. Sci.,2006

4. Adenosine A2A receptor blockade prevents synaptotoxicity and memory dysfunction caused by beta-amyloid peptides via p38 mitogen-activated protein kinase pathway;Canas;J. Neurosci.,2009

5. Alzheimer amyloid beta-peptide inhibits the late phase of long-term potentiation through calcineurin-dependent mechanisms in the hippocampal dentate gyrus;Chen;Neurobiol. Learn. Mem.,2002

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