Pathogenic potential of human SLC12A5 variants causing KCC2 dysfunction-Reference-Cited by-同舟云学术

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Pathogenic potential of human SLC12A5 variants causing KCC2 dysfunction

Published:2019-05 Issue: Volume:1710 Page:1-7
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Fukuda Atsuo^ORCID,Watanabe Miho

Funder

Grants-in-Aid for Scientific Research on Innovative Areas

Ministry of Education, Culture, Sports, Science and Technology of Japan

Grants-in-Aid for Scientific Research

Challenging Research

Japan Society for the Promotion of Science

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference53 articles.

1. Hyperpolarizing GABAergic transmission requires the KCC2 C-terminal ISO domain;Acton;J. Neurosci.,2012

2. Molecular architecture of potassium chloride co-transporter KCC2;Agez;Sci. Rep.,2017

3. Excitatory actions of GABA during development: the nature of the nurture;Ben-Ari;Nat. Rev. Neurosci.,2002

4. Oligomerization of KCC2 correlates with development of inhibitory neurotransmission;Blaesse;J. Neurosci.,2006

5. Epilepsy as a neurodevelopmental disorder;Bozzi;Front. Psychiatry,2012

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