TBHQ improved neurological recovery after traumatic brain injury by inhibiting the overactivation of astrocytes-Reference-Cited by-同舟云学术

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TBHQ improved neurological recovery after traumatic brain injury by inhibiting the overactivation of astrocytes

Published:2020-07 Issue: Volume:1739 Page:146818
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Zhang Zhen-Wen,Liang Jun,Yan Jing-Xing,Ye Yi-Chao,Wang Jing-Jing,Chen Chong,Sun Hong-Tao,Chen Feng,Tu Yue,Li Xiao-Hong

Funder

National Nature Scientific Fund of China

Nature Scientific Fund of Tianjin

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference65 articles.

1. Contuse lesion of the rat spinal cord of moderate intensity leads to a higher time-dependent secondary neurodegeneration than severe one. An open-window for experimental neuroprotective interventions;Andrade;Tissue Cell,2008

2. Tert-butylhydroquinone attenuates oxidative stress and inflammation in hypothalamic paraventricular nucleus in high salt-induced hypertension;Bai;Toxicol. Lett.,2017

3. A CD11d monoclonal antibody treatment reduces tissue injury and improves neurological outcome after fluid percussion brain injury in rats;Bao;J. Neurotrauma,2012

4. Inhibition of astroglial nuclear factor kappaB reduces inflammation and improves functional recovery after spinal cord injury;Brambilla;J. Exp. Med.,2005

5. Transgenic inhibition of astroglial NF-kappa B improves functional outcome in experimental autoimmune encephalomyelitis by suppressing chronic central nervous system inflammation;Brambilla;J. Immunol.,2009

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