Mice lacking spinal α2GABAA receptors: Altered GABAergic neurotransmission, diminished GABAergic antihyperalgesia, and potential compensatory mechanisms preventing a hyperalgesic phenotype-Reference-Cited by-同舟云学术

Mice lacking spinal α2GABAA receptors: Altered GABAergic neurotransmission, diminished GABAergic antihyperalgesia, and potential compensatory mechanisms preventing a hyperalgesic phenotype

Published:2020-08 Issue: Volume:1741 Page:146889
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Tudeau Laetitia,Acuña Mario A.,Albisetti Gioele W.,Neumann Elena,Ralvenius William T.,Scheurer Louis,Poe Michael,Cook James M.,Johannssen Helge C.,Zeilhofer Hanns Ulrich

Funder

Swiss National Science Foundation to HUZ

National Institutes of Health to JMC

Milwaukee Institute of Drug Discovery

Deutsche Forschungsgemeinschaft

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference64 articles.

1. α4β3δ GABAA receptors characterized by fluorescence resonance energy transfer-derived measurements of membrane potential;Adkins;J. Biol. Chem.,2001

2. PGE2 selectively blocks inhibitory glycinergic neurotransmission onto rat superficial dorsal horn neurons;Ahmadi;Nat. Neurosci.,2002