Piriform cortex alterations in the Ts65Dn model for down syndrome
Author:
Funder
Jerome Lejeune Foundation
Ministry of Science, Innovation and Universities
Publisher
Elsevier BV
Subject
Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience
Reference76 articles.
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2. Dendritic atrophy in children with Down’s syndrome;Becker;Ann. Neurol.,1986
3. The “Down syndrome critical region” is sufficient in the mouse model to confer behavioral, neurophysiological, and synaptic phenotypes characteristic of Down syndrome;Belichenko;J. Neurosci.,2009
4. Excitatory-inhibitory relationship in the fascia dentata in the Ts65Dn mouse model of down syndrome;Belichenko;J. Comp. Neurol.,2009
5. Synaptic and cognitive abnormalities in mouse models of Down syndrome: Exploring genotype-phenotype relationships;Belichenko;J. Comp. Neurol.,2007
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1. Early chronic fasudil treatment rescues hippocampal alterations in the Ts65Dn model for down syndrome;Neurochemistry International;2024-03
2. Basal forebrain cholinergic neurons are vulnerable in a mouse model of Down syndrome and their molecular fingerprint is rescued by maternal choline supplementation;The FASEB Journal;2023-05-16
3. Brain circuit pathology in Down syndrome: from neurons to neural networks;Reviews in the Neurosciences;2022-09-29
4. Social defeat drives hyperexcitation of the piriform cortex to induce learning and memory impairment but not mood-related disorders in mice;Translational Psychiatry;2022-09-10
5. Cholinergic Senescence in the Ts65Dn Mouse Model for Down Syndrome;Neurochemical Research;2022-06-29
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