Inhaled nitric oxide protects cerebral autoregulation through prevention of impairment of ATP and calcium sensitive K channel mediated cerebrovasodilation after traumatic brain injury-Reference-Cited by-同舟云学术

Inhaled nitric oxide protects cerebral autoregulation through prevention of impairment of ATP and calcium sensitive K channel mediated cerebrovasodilation after traumatic brain injury

Published:2019-05 Issue: Volume:1711 Page:1-6
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Pastor Philip,Curvello Victor,Hekierski Hugh,Armstead William M.

Funder

Mallinckrodt Pharmaceuticals

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference19 articles.

1. Brain injury impairs ATP-sensitive K+ channel function in piglet cerebral arteries;Armstead;Stroke,1997

2. Hypotension dilates pial arteries by KATP and Kca channel activation;Armstead;Brain Res.,1999

3. Endothelins and the role of endothelin antagonists and the management of posttraumatic vasospasm;Armstead;Curr. Pharm. Des.,2004

4. Cerebral blood flow autoregulation and dysregulation;Armstead;Anesthesiol. Clin.,2016

5. SNP improves cerebral hemodynamics during normotension but fails to prevent sex dependent impaired cerebral autoregulation during hypotension after brain injury;Armstead;Brain Res.,2010

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