Non-cell autonomous mechanism of Parkinson’s disease pathology caused by G2019S LRRK2 mutation in Ashkenazi Jewish patient: Single cell analysis-Reference-Cited by-同舟云学术

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Non-cell autonomous mechanism of Parkinson’s disease pathology caused by G2019S LRRK2 mutation in Ashkenazi Jewish patient: Single cell analysis

Published:2019-11 Issue: Volume:1722 Page:146342
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Kim Jeffrey,Daadi Marcel M.

Funder

Robert J. Kleberg, Jr. and Helen C. Kleberg Foundation

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference88 articles.

1. Developmentally regulated expression of Neuro-p24 and its possible function in neurite extension;Araki;Neurosci. Res.,2002

2. Mitochondrial dysfunction in the limelight of Parkinson's disease pathogenesis;Banerjee;Biochim. Biophys. Acta,2009

3. Unbiased screen for interactors of leucine-rich repeat kinase 2 supports a common pathway for sporadic and familial Parkinson disease;Beilina;Proc. Natl. Acad. Sci.,2014

4. Gene expression profiling in single cells from the pancreatic islets of Langerhans reveals lognormal distribution of mRNA levels;Bengtsson;Genome Res.,2005

5. Mitochondrial complex I deficiency caused by a deleterious NDUFA11 mutation;Berger;Ann. Neurol.,2008

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3. LRRK2 Attenuates Antioxidant Response in Familial Parkinson’s Disease Derived Neural Stem Cells;Cells;2023-10-31

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