Electrophysiological characterization of substantia nigra dopaminergic neurons in partially lesioned rats: Effects of subthalamotomy and levodopa treatment-Reference-Cited by-同舟云学术

Electrophysiological characterization of substantia nigra dopaminergic neurons in partially lesioned rats: Effects of subthalamotomy and levodopa treatment

Published:2006-04 Issue:1 Volume:1084 Page:175-184
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Bilbao Gaizka,Ruiz-Ortega Jose Angel,Miguens Natalia,Ulibarri Isabel,Linazasoro Gurutz,Gómez-Urquijo Sonia,Garibi Jesús,Ugedo Luisa

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference44 articles.

1. Hyperactivity of remaining dopaminergic neurones after partial destruction of the nigro-striatal dopaminergic system in the rat;Agid;Nat. New Biol.,1973

2. Inhibition of nigral dopamine neurons by systemic and local apomorphine: possible contribution of dendritic autoreceptors;Akaoka;Neuroscience,1992

3. Bilateral subthalamotomy in Parkinson's disease: initial and long-term response;Alvarez;Brain,2005

4. Microrecordings of subthalamic nucleus and substantia nigra pars reticulata neuronal activity in parkinsonian patients;Benazzouz;Mov. Disord.,1996

5. The primate subthalamic nucleus. II. Neuronal activity in the MPTP model of parkinsonism;Bergman;J. Neurophysiol.,1994

Cited by 27 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration;2024-07-26

2. Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration;2024-07-26

3. GLP-1 modulated the firing activity of nigral dopaminergic neurons in both normal and parkinsonian mice;Neuropharmacology;2024-07

4. Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration;2024-04-10

5. Role of dopamine neuron activity in Parkinson's disease pathophysiology;Experimental Neurology;2024-03