Subchronic administration of l-DOPA to adult rats with a unilateral 6-hydroxydopamine lesion of dopamine neurons results in a sensitization of enhanced GABA release in the substantia nigra, pars reticulata
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience
Reference23 articles.
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2. Activation of D1 receptors stimulates accumulation of gamma-aminobutyric acid in slices of the pars reticulata of 6-hydroxydopamine-lesioned rats;Aceves;Neurosci. Lett.,1992
3. Serotonin–GABA interactions modulate MDMA-induced mesolimbic dopamine release;Bankson;J. Neurochem.,2004
4. Effects of AMPA and D1 receptor activation on striatal and nigral GABA efflux;Byrnes;Syanpse,1997
5. Selective modifications in GAD67 mRNA levels in striatonigral and striatopallidal pathways correlate to dopamine agonist priming in 6-hydroxydopamine-lesioned rats;Carta;Eur. J. Neurosci.,2003
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