HDAC inhibitor ameliorates behavioral deficits in Mecp2308/y mouse model of Rett syndrome-Reference-Cited by-同舟云学术

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HDAC inhibitor ameliorates behavioral deficits in Mecp2308/y mouse model of Rett syndrome

Published:2021-12 Issue: Volume:1772 Page:147670
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Lebrun Nicolas,Delépine Chloé,Selloum Mohamed,Meziane Hamid,Nectoux Juliette,Herault Yann,Bienvenu Thierry^ORCID

Funder

National Institutes of Health

Agence Nationale de la Recherche

Fondation Jérôme Lejeune

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference55 articles.

1. Presymptomatic training mitigates functional deficits in a mouse model of Rett syndrome;Achilly;Nature,2021

2. Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2;Amir;Nat. Genet.,1999

3. Inhibition of HDAC6 deacetylase activity increases its binding with microtubules and suppresses microtubule dynamic instability in MCF-7 cells;Asthana;J. Biol. Chem.,2013

4. Non-cell autonomous influence of MeCP2-deficient glia on neuronal dendritic morphology;Ballas;Nat. Neurosci.,2009

5. MECP2 missense mutations outside the canonical MBD and TRD domains in males with intellectual disability;Bianciardi;J. Hum. Genet.,2016

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