Neuroprotection by erythropoietin administration after experimental traumatic brain injury

Author:

Grasso Giovanni,Sfacteria Alessandra,Meli Francesco,Fodale Vincenzo,Buemi Michele,Iacopino Domenico G.

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference50 articles.

1. 2007. Rates of hospitalization related to traumatic brain injury-nine states, 2003. MMWR Morb Mortal Wkly Rep. 56, 167–70.

2. Modulation of aquaporin-4 water transport in a model of TBI;Amorini;Acta Neurochir., Suppl.,2003

3. Protein kinase B (c-Akt), phosphatidylinositol 3-kinase, and STAT5 are activated by erythropoietin (EPO) in HCD57 erythroid cells but are constitutively active in an EPO-independent, apoptosis-resistant subclone (HCD57-SREI cells);Bao;Blood,1999

4. Erythropoietin crosses the blood–brain barrier to protect against experimental brain injury;Brines;Proc. Natl. Acad. Sci. U. S. A.,2000

5. Recombinant human erythropoietin (rHuEPO): more than just the correction of uremic anemia;Buemi;J. Nephrol.,2002

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