Dihydroartemisinin inhibits the proliferation of IgAN mesangial cells through the mTOR signaling pathway-Reference-Cited by-同舟云学术

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Dihydroartemisinin inhibits the proliferation of IgAN mesangial cells through the mTOR signaling pathway

Published:2020-03 Issue: Volume:80 Page:106125
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Xia Ming,Liu Di,Tang Xiaofang,Liu Yexin,Liu Haiyang,Liu Yu,Chen Guochun,Liu Hong

Funder

National Natural Science Foundation of China

Central South University in China

Publisher

Elsevier BV

Subject

Pharmacology,Immunology,Immunology and Allergy

Reference36 articles.

1. IgA nephropathy;Lai;Nat. Rev. Dis. Primers,2016

2. Individuals of Pacific Asian origin with IgA nephropathy have an increased risk of progression to end-stage renal disease;Barbour;Kidney Int.,2013

3. mTOR signaling in growth, metabolism, and disease;Saxton;Cell,2017

4. Rapamycin enhances repressed autophagy and attenuates aggressive progression in a rat model of IgA nephropathy;Liu;Am. J. Nephrol.,2017

5. Pathogenic significance of IgA receptor interactions in IgA nephropathy;Monteiro;Trends Mol. Med.,2002

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1. Comprehensive Analysis of ceRNA Network and Immune Cell Infiltration Pattern of Autophagy-Related Genes in IgA Nephropathy;Kidney and Blood Pressure Research;2024-05-31

2. Artemisinin and its derivatives as promising therapies for autoimmune diseases;Heliyon;2024-04

3. Traditional Chinese medicine for the treatment of immune-related nephropathy: A review;Acta Pharmaceutica Sinica B;2024-01

4. Artemisinins: Promising drug candidates for the treatment of autoimmune diseases;Medicinal Research Reviews;2023-12-06

5. EIF2α/ATF4 pathway enhances proliferation of mesangial cell via cyclin D1 during endoplasmic reticulum stress in IgA nephropathy;Clinical Immunology;2023-12

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