Promotion of Raf-1/ASK1 complex formation by corylin inhibits cell apoptosis in myocardial ischemia/reperfusion injury-Reference-Cited by-同舟云学术

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Promotion of Raf-1/ASK1 complex formation by corylin inhibits cell apoptosis in myocardial ischemia/reperfusion injury

Published:2024-10 Issue: Volume:140 Page:112921
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Huang Kaiyu,Cai Chenchen,He Hualing,Yi Binghua,Xu Wencai,Lin Zhonghao,Lv Xiao,Liu Ronghua^ORCID,Zheng Cheng,Zhou Yingying,Lin Jiafeng^ORCID

Funder

National Natural Science Foundation of China

Wenzhou Municipal Science and Technology Bureau

Scientific Research Foundation of Education Department of Anhui Province of China

Zhejiang Province Natural Science Foundation

Henan Provincial Science and Technology Research Project

National Natural Science Foundation of China-Yunnan Joint Fund

National Aerospace Science Foundation of China

Publisher

Elsevier BV

Reference28 articles.

1. The 10th Biennial Hatter Cardiovascular Institute workshop: cellular protection-evaluating new directions in the setting of myocardial infarction, ischaemic stroke, and cardio-oncology;Davidson;Basic Res. Cardiol.,2018

2. Myocardial repair/remodelling following infarction: roles of local factors;Sun;Cardiovasc. Res.,2009

3. Raf-1 promotes cell survival by antagonizing apoptosis signal-regulating kinase 1 through a MEK-ERK independent mechanism;Chen;PNAS,2001

4. Meaningful relationships: the regulation of the Ras/Raf/MEK/ERK pathway by protein interactions;Kolch;Biochem. J.,2000

5. C. Changmin, S. Arthur J, Apoptosis-linked gene-2 connects the Raf-1 and ASK1 signalings, Biochemical and biophysical research communications 333(1) (2005).

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