The role of HMGB1 on TDI-induced NLPR3 inflammasome activation via ROS/NF-κB pathway in HBE cells-Reference-Cited by-同舟云学术

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The role of HMGB1 on TDI-induced NLPR3 inflammasome activation via ROS/NF-κB pathway in HBE cells

Published:2021-09 Issue: Volume:98 Page:107859
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Jiao Bo,Guo Sumei,Yang Xiaohan,Sun Lei,Sai Linlin,Yu Gongchang,Bo Cunxiang,Zhang Yu,Peng Cheng,Jia Qiang,Dai Yufei

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Pharmacology,Immunology,Immunology and Allergy

Reference44 articles.

1. Extracellular HMGB1 as a therapeutic target in inflammatory diseases;Andersson;Expert. Opin. Ther. Targets.,2018

2. Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression;Bauernfeind;J. Immunol.,2009

3. NLRP3 inflammasome is required in murine asthma in the absence of aluminum adjuvant;Besnard;Allergy,2011

4. Evaluation of anti-asthmatic and antioxidant potential of Boerhavia procumbens in toluene diisocyanate (TDI) treated rats;Bokhari;J. Ethnopharmacol.,2015

5. Role of clusterin/progranulin in toluene diisocyanate-induced occupational asthma;Choi;Exp. Mol. Med.,2018

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