Activation of IRF3 contributes to IFN-γ and ISG54 expression during the immune responses to B16F10 tumor growth-Reference-Cited by-同舟云学术

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Activation of IRF3 contributes to IFN-γ and ISG54 expression during the immune responses to B16F10 tumor growth

Published:2017-09 Issue: Volume:50 Page:121-129
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Guinn Zachary,Brown Deborah M.,Petro Thomas M.

Funder

University of Nebraska Medical Center

University of Nebraska-Lincoln

National Institutes of Health

Publisher

Elsevier BV

Subject

Pharmacology,Immunology,Immunology and Allergy

Reference33 articles.

1. Virus infection induces the assembly of coordinately activated transcription factors on the IFN-beta enhancer in vivo;Wathelet;Mol. Cell,1998

2. Aberrant expression of interferon regulatory factor 3 in human lung cancer;Tokunaga;Biochem. Biophys. Res. Commun.,2010

3. Toll-like receptor 3 agonist complexed with cationic liposome augments vaccine-elicited antitumor immunity by enhancing TLR3-IRF3 signaling and type I interferons in dendritic cells;Wang;Vaccine,2012

4. Interferon response factor 3 is crucial to poly-I:C induced NK cell activity and control of B16 melanoma growth;Moore;Cancer Lett.,2014

5. Significant role for IRF3 in both T cell and APC effector functions during T cell responses;Guinn;Cell. Immunol.,2016

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