Stimulation of nuclear receptor REV-ERBs alleviates monosodium iodoacetate-induced osteoarthritis pathology of mice and the induction of inflammatory molecules expression in primary cultured chondrocytes-Reference-Cited by-同舟云学术

Stimulation of nuclear receptor REV-ERBs alleviates monosodium iodoacetate-induced osteoarthritis pathology of mice and the induction of inflammatory molecules expression in primary cultured chondrocytes

Published:2024-01 Issue: Volume:127 Page:111349
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Hashizume Hiroki,Motonari Hatsune,Yamamoto Kenta,Nakamura Yoki,Hisaoka-Nakashima Kazue,Morioka Norimitsu^ORCID

Funder

Hiroshima University

Takeda Science Foundation

Natural Science Center for Basic Research and Development, Hiroshima University

Nakatomi Foundation

Suzuken Memorial Foundation

Uehara Memorial Foundation

Publisher

Elsevier BV

Subject

Pharmacology,Immunology,Immunology and Allergy

Reference15 articles.

1. Persistent pain after joint replacement: prevalence, sensory qualities, and postoperative determinants;Wylde;Pain,2011

2. Evaluation of intracellular signal molecules that regulate TLR4-stimulated inflammatory mediator expression in cultured rat chondrocytes;Hashizume;J. Pharmacol. Sci.,2023

3. Stimulation of nuclear receptor REV-ERBs suppresses inflammatory responses in spinal microglia;Morioka;Neurochem. Int.,2021

4. Gene regulation. Discrete functions of nuclear receptor Rev-erbα couple metabolism to the clock;Zhang;Science,2015

5. Stimulation of nuclear receptor REV-ERBs suppresses production of pronociceptive molecules in cultured spinal astrocytes and ameliorates mechanical hypersensitivity of inflammatory and neuropathic pain of mice;Morioka;Brain. Behav. Immun.,2019

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1. AFK-PD alleviated osteoarthritis progression by chondroprotective and anti-inflammatory activity;Frontiers in Pharmacology;2024-08-29