CFTR Therapeutics Normalize Cerebral Perfusion Deficits in Mouse Models of Heart Failure and Subarachnoid Hemorrhage-Reference-Cited by-同舟云学术

CFTR Therapeutics Normalize Cerebral Perfusion Deficits in Mouse Models of Heart Failure and Subarachnoid Hemorrhage

Published:2019-12 Issue:8 Volume:4 Page:940-958
ISSN:2452-302X
Container-title:JACC: Basic to Translational Science
language:en
Short-container-title:JACC: Basic to Translational Science

Author:

Lidington Darcy,Fares Jessica C.,Uhl Franziska E.,Dinh Danny D.,Kroetsch Jeffrey T.,Sauvé Meghan,Malik Firhan A.,Matthes Frank,Vanherle Lotte,Adel Arman,Momen Abdul,Zhang Hangjun,Aschar-Sobbi Roozbeh,Foltz Warren D.,Wan Hoyee,Sumiyoshi Manabu,Macdonald R. Loch,Husain Mansoor,Backx Peter H.,Heximer Scott P.,Meissner Anja,Bolz Steffen-Sebastian

Funder

Natural Sciences and Engineering Research Council of Canada

Ontario Council on Graduate Studies, Council of Ontario Universities

Brain Aneurysm Foundation

Canadian Institutes of Health Research

Knut och Alice Wallenbergs Stiftelse

Heart and Stroke Foundation of Canada

Royal Swedish Academy of Sciences

Canada Research Chairs

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference31 articles.

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3. Evaluation of cognitive impairment by the Montreal cognitive assessment in patients with aneurysmal subarachnoid haemorrhage: prevalence, risk factors and correlations with 3 month outcomes;Wong;J Neurol Neurosurg Psychiatry,2012

4. Proximal cerebral arteries develop myogenic responsiveness in heart failure via tumor necrosis factor-α-dependent activation of sphingosine-1-phosphate signaling;Yang;Circulation,2012

5. Tumor necrosis factor-α underlies loss of cortical dendritic spine density in a mouse model of congestive heart failure;Meissner;J Am Heart Assoc,2015

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