Response gene to complement 32 is required for C5b-9 induced cell cycle activation in endothelial cells

Author:

Fosbrink Matthew,Cudrici Cornelia,Tegla Cosmin A.,Soloviova Kateryna,Ito Takahiro,Vlaicu Sonia,Rus Violeta,Niculescu Florin,Rus Horea

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Molecular Biology,Pathology and Forensic Medicine

Reference34 articles.

1. RGC-32 increases p34CDC2 kinase activity and entry of aortic smooth muscle cells into S-phase;Badea;J. Biol. Chem.,2002

2. Molecular cloning and characterization of RGC-32, a novel gene induced by complement activation in oligodendrocytes;Badea;J. Biol. Chem.,1998

3. Endothelial cell-to-cell junctions: molecular organization and role in vascular homeostasis;Bazzoni;Physiol. Rev.,2004

4. Terminal complement proteins C5b-9 release basic fibroblast growth factor and platelet-derived growth factor from endothelial cells;Benzaquen;J. Exp. Med.,1994

5. Elimination of terminal complement intermediates from the plasma membrane of nucleated cells: the rate of disappearance differs for cells carrying C5b-7 or C5b-8 or a mixture of C5b-8 with a limited number of C5b-9;Carney;J. Immunol.,1985

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