Preoptic recess lesions reduce right atrial pressure responses to volume expansion

Author:

Bealer Steven L.

Publisher

Elsevier BV

Subject

Neurology (clinical),Physiology,General Neuroscience

Reference26 articles.

1. Cardiac output and renal excretion rates during acute blood volume expansion in rats;Ackermann;Am. J. Physiol. (Heart Circ. Physiol.),1978

2. Control of renal function in isovolemic hemodilution or in vagotomized, infused rats;Ackermann;Pfluegers Arch.,1980

3. Control of right atrial pressure at constant cardiac output suppresses volume natriuresis in anesthetized rats;Ackermann;Can. J. Physiol. Pharmacol.,1984

4. Hemodynamic mechanisms in CNS-induced natriuresis in the conscious rat;Bealer;Am. J. Physiol,1983

5. Acute hypertensive and natriuretic responses following preoptic hypothalamic lesions;Bealer;Am. J. Med. Sci.,1988

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1. Modulation of noradrenaline release in the median preoptic area by GABAergic inputs from the organum vasculosum of the lamina terminalis in the rat;Neurochemistry International;2004-02

2. Systemic angiotensin II and volume expansion release norepinephrine in the preoptic recess;Brain Research;2000-05

3. Central control of cardiac baroreflex responses during peripheral hyperosmolality;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;2000-05-01

4. Osmolality: a physiological long-term regulator of lumbar sympathetic nerve activity and arterial pressure;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;1999-06-01

5. Preoptic recess alpha-adrenoceptors control cardiovascular responses to hyperosmolality;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;1997-04-01

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