Progastrin production transitions from Bmi1+/Prox1+ to Lgr5high cells during early intestinal tumorigenesis-Reference-Cited by-同舟云学术

Progastrin production transitions from Bmi1+/Prox1+ to Lgr5high cells during early intestinal tumorigenesis

Published:2021-02 Issue:2 Volume:14 Page:101001
ISSN:1936-5233
Container-title:Translational Oncology
language:en
Short-container-title:Translational Oncology

Author:

Giraud J.,Foroutan M.,Boubaker-Vitre J.,Grillet F.,Homayed Z.^ORCID,Jadhav U.,Crespy P.,Breuker C.,Bourgaux J-F.,Hazerbroucq J.,Pignodel C.,Brulin B.^ORCID,Shivdasani R.A.^ORCID,Jay P.^ORCID,Hollande F.^ORCID,Pannequin J.

Funder

Australian NH&MRC

SIRIC Montpellier Cancer

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference44 articles.

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2. Beta-catenin/Tcf-4 inhibition after progastrin targeting reduces growth and drives differentiation of intestinal tumors;Pannequin;Gastroenterology,2007

3. Gastrin gene expression is required for the proliferation and tumorigenicity of human colon cancer cells;Singh;Cancer Res.,1996

4. Gastrin is a target of the beta-catenin/TCF-4 growth-signaling pathway in a model of intestinal polyposis;Koh;J. Clin. Investig.,2000

5. Activation of beta-catenin-Tcf signaling in colon cancer by mutations in beta-catenin or APC;Morin;Science,1997

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