Accelerated transmural gradients of energy compound metabolism resulting from left ventricular hypertrophy-Reference-Cited by-同舟云学术

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Accelerated transmural gradients of energy compound metabolism resulting from left ventricular hypertrophy

Published:1990-10 Issue:4 Volume:100 Page:506-516
ISSN:0022-5223
Container-title:The Journal of Thoracic and Cardiovascular Surgery
language:en
Short-container-title:The Journal of Thoracic and Cardiovascular Surgery

Author:

Bladergroen Mark R.,Takei Hiroshi,Christopher Thomas D.,Cummings Robin G.,Blanchard Susan M.,Lowe James E.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Pulmonary and Respiratory Medicine,Surgery

Reference48 articles.

1. Ischemic contracture of the heart: “stone heart.”;Cooley;Am J Cardiol,1972

2. Normothermic myocardial anoxia: effects on the canine heart with left ventricular outflow obstruction;Levitsky;Ann Thorac Surg,1971

3. Characteristics of chronic left ventricular hypertrophy induced by subcoronary valvular aortic stenosis. II. Response to ischemia;Attarian;J Thorac Cardiovasc Surg,1981

4. Response of hypertrophied myocardium to ischemia: correlation with biochemical and physiological parameters;Sink;J Thorac Cardiovasc Surg,1981

5. Depressed high-energy phosphate content in hypertrophied ventricles of animals and man;Peyton;J Thorac Cardiovasc Surg,1982

Cited by 11 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Regulation of H9C2 cell hypertrophy by 14-3-3η via inhibiting glycolysis;PLOS ONE;2024-07-22

2. AORTIC ARCH REPAIR WITH CORONARY ARTERY REVASCULARIZATION;FUKUSHIMA JOURNAL OF MEDICAL SCIENCE;2009

3. Impaired activation of ATP-sensitive K+channels in endocardial myocytes from left ventricular hypertrophy;American Journal of Physiology-Heart and Circulatory Physiology;2007-12

4. Increased susceptibility of hypertrophied hearts to ischemic injury;The Annals of Thoracic Surgery;2003-02

5. Mitochondrial Energy Metabolism in the Left Ventricular Tissue of Spontaneously Hypertensive Rats: Abnormalities in both Adeninenucleotide and Phosphate Translocators and Enzyme Adenylate-Kinase and Creatine-Phosphokinase Activities;Clinical and Experimental Hypertension;1998-01

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