Basal nitric oxide expresses endogenous cardioprotection during reperfusion by inhibition of neutrophil-mediated damage after surgical revascularization-Reference-Cited by-同舟云学术

Basal nitric oxide expresses endogenous cardioprotection during reperfusion by inhibition of neutrophil-mediated damage after surgical revascularization

Published:1997-02 Issue:2 Volume:113 Page:399-409
ISSN:0022-5223
Container-title:The Journal of Thoracic and Cardiovascular Surgery
language:en
Short-container-title:The Journal of Thoracic and Cardiovascular Surgery

Author:

Sato Hiroki,Zhao Zhi-Qing,Jordan James E.,Todd James C.,Riley Robert D.,Taft C.Spencer,Hammon Jr John W.,Li Ping,Ma Xin-liang,Vinten-Johansen J.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Pulmonary and Respiratory Medicine,Surgery

Reference20 articles.

1. Diminished basal nitric oxide release after myocardial ischemia and reperfusion promotes neutrophil adherence to coronary endothelium;Ma;Circ Res,1993

2. Reperfusion after acute coronary occlusion in dogs impairs endothelium-dependent relaxation to acetylcholine and augments contractile reactivity in vitro;VanBenthuysen;J Clin Invest,1987

3. Time course of endothelial dysfunction and myocardial injury during myocardial ischemia and reperfusion in the cat;Tsao;Circulation,1990

4. Intracoronary L-arginine during reperfusion improves endothelial function and reduces infarct size;Nakanishi;Am J Physiol,1992

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