Phospholipase Cγ in Toll-like receptor-mediated inflammation and innate immunity-Reference-Cited by-同舟云学术

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Phospholipase Cγ in Toll-like receptor-mediated inflammation and innate immunity

Published:2017-01 Issue: Volume:63 Page:92-97
ISSN:2212-4926
Container-title:Advances in Biological Regulation
language:en
Short-container-title:Advances in Biological Regulation

Author:

Bae Yoe-Sik,Lee Ha Young,Jung Young Su,Lee Mingyu,Suh Pann-Ghill

Funder

National Research Foundation of Korea

Ministry of Science, ICT and future Planning

Publisher

Elsevier BV

Subject

Cancer Research,Genetics,Molecular Biology,Molecular Medicine

Reference65 articles.

1. TLR-mediated activation of NK cells and their role in bacterial/viral immune responses in mammals;Adib-Conquy;Immunol. Cell Biol.,2014

2. The p110δ isoform of the kinase PI(3)K controls the subcellular compartmentalization of TLR4 signaling and protects from endotoxic shock;Aksoy;Nat. Immunol.,2012

3. Macrophages generate reactive oxygen species in response to minimally oxidized low-density lipoprotein: toll-like receptor 4- and spleen tyrosine kinase-dependent activation of NADPH oxidase 2;Bae;Circ. Res.,2009

4. Localized biphasic changes in phosphatidylinositol-4,5-bisphosphate at sites of phagocytosis;Botelho;J. Cell. Biol.,2000

5. Phospholipase C-gamma2 is essential for NK cell cytotoxicity and innate immunity to malignant and virally infected cells;Caraux;Blood,2006

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