PLCγ1: Potential arbitrator of cancer progression-Reference-Cited by-同舟云学术

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PLCγ1: Potential arbitrator of cancer progression

Published:2018-01 Issue: Volume:67 Page:179-189
ISSN:2212-4926
Container-title:Advances in Biological Regulation
language:en
Short-container-title:Advances in Biological Regulation

Author:

Jang Hyun-Jun,Suh Pann-Ghill,Lee Yu Jin,Shin Kyeong Jin,Cocco Lucio,Chae Young Chan

Funder

National Research Foundation of Korea

NRF Bio & Medical Technology Development Program

MSIP

MOHW

Publisher

Elsevier BV

Subject

Cancer Research,Genetics,Molecular Biology,Molecular Medicine

Reference93 articles.

1. PLC-gamma activation is required for PDGF-betaR-mediated mitogenesis and monocytic differentiation of myeloid progenitor cells;Alimandi;Oncogene,1997

2. Defining the stoichiometry of inositol 1,4,5-trisphosphate binding required to initiate Ca2+ release;Alzayady;Sci. Signal,2016

3. IRBIT, a novel inositol 1,4,5-trisphosphate (IP3) receptor-binding protein, is released from the IP3 receptor upon IP3 binding to the receptor;Ando;J. Biol. Chem.,2003

4. Elevated content of the tyrosine kinase substrate phospholipase C-gamma 1 in primary human breast carcinomas;Arteaga;Proc. Natl. Acad. Sci. U. S. A.,1991

5. Kaposi's sarcoma herpesvirus K15 protein contributes to virus-induced angiogenesis by recruiting PLCgamma1 and activating NFAT1-dependent RCAN1 expression;Bala;PLoS Pathog.,2012

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