PI3K signaling in the pathogenesis of obesity: The cause and the cure-Reference-Cited by-同舟云学术

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PI3K signaling in the pathogenesis of obesity: The cause and the cure

Published:2015-05 Issue: Volume:58 Page:1-15
ISSN:2212-4926
Container-title:Advances in Biological Regulation
language:en
Short-container-title:Advances in Biological Regulation

Author:

Beretta Martina,Bauer Michael,Hirsch Emilio^ORCID

Funder

Telethon

Cassa di Risparmio di Torino and Leducq Foundation

Jena School for Microbial Communication (JSMC)

Center for Sepsis Control and Care (CSCC)

Publisher

Elsevier BV

Subject

Cancer Research,Genetics,Molecular Biology,Molecular Medicine

Reference147 articles.

1. The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307);Aguirre;J Biol Chem,2000

2. Phosphorylation of Ser307 in insulin receptor substrate-1 blocks interactions with the insulin receptor and inhibits insulin action;Aguirre;J Biol Chem,2002

3. Dominant role of the p110beta isoform of PI3K over p110alpha in energy homeostasis regulation by POMC and AgRP neurons;Al-Qassab;Cell Metab,2009

4. Molecular basis for the substrate specificity of protein kinase B; comparison with MAPKAP kinase-1 and p70 S6 kinase;Alessi;FEBS Lett,1996

5. Isoform-specific regulation of insulin-dependent glucose uptake by Akt/protein kinase B;Bae;J Biol Chem,2003

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