The HIV-Tat protein interacts with Sp3 transcription factor and inhibits its binding to a distal site of the sod2 promoter in human pulmonary artery endothelial cells-Reference-Cited by-同舟云学术

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The HIV-Tat protein interacts with Sp3 transcription factor and inhibits its binding to a distal site of the sod2 promoter in human pulmonary artery endothelial cells

Published:2020-02 Issue: Volume:147 Page:102-113
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Manes Terrin L.,Simenauer Ari,Geohring Jason L.,Flemming Juliana,Brehm Michael,Cota-Gomez Adela

Funder

National Institutes of Health

National Heart, Lung and Blood Institute

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference44 articles.

1. Non-infectious pulmonary diseases and HIV;Triplette;Curr. HIV AIDS Rep.,2016

2. Oxidative stress during HIV infection: mechanisms and consequences;Ivanov;Oxid. Med. Cell. Longev.,2016

3. Tat protein of human immunodeficiency virus type 1 represses expression of manganese superoxide dismutase in HeLa cells;Flores;Proc. Natl. Acad. Sci. U. S. A,1993

4. HIV-1 tat protein tram-activates transcription in vitro;Marciniak;Cell,1990

5. HIV-1 Tat protein promotes formation of more-processive elongation complexes;Marciniak;EMBO J.,1991

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The Intersection of HIV and Pulmonary Vascular Health: From HIV Evolution to Vascular Cell Types to Disease Mechanisms;Journal of Vascular Diseases;2024-05-06

2. The DNA Damage Response and HIV-Associated Pulmonary Arterial Hypertension;International Journal of Molecular Sciences;2020-05-07

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