Selective binding of nuclear alpha-synuclein to the PGC1alpha promoter under conditions of oxidative stress may contribute to losses in mitochondrial function: Implications for Parkinson’s disease-Reference-Cited by-同舟云学术

Selective binding of nuclear alpha-synuclein to the PGC1alpha promoter under conditions of oxidative stress may contribute to losses in mitochondrial function: Implications for Parkinson’s disease

Published:2012-08 Issue:4 Volume:53 Page:993-1003
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Siddiqui Almas,Chinta Shankar J.,Mallajosyula Jyothi K.,Rajagopolan Subramanian,Hanson Ingrid,Rane Anand,Melov Simon,Andersen Julie K.

Funder

R01 NS045615 (J.K.A.), Allison Foundation

National Parkinson's Foundation (J.K.M.)

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

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3. Redox reactions of the alpha-synuclein-Cu(2+) complex and their effects on neuronal cell viability;Wang;Biochemistry,2010

4. A53T-alpha-synuclein overexpression impairs dopamine signaling and striatal synaptic plasticity in old mice;Kurz;PLoS One,2010

5. Elevated tauopathy and alpha-synuclein pathology in postmortem Parkinson's disease brains with and without dementia;Wills;Exp. Neurol,2010

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