Redox cycling and increased oxygen utilization contribute to diquat-induced oxidative stress and cytotoxicity in Chinese hamster ovary cells overexpressing NADPH-cytochrome P450 reductase

Author:

Fussell Karma C.,Udasin Ronald G.,Gray Joshua P.,Mishin Vladimir,Smith Peter J.S.,Heck Diane E.,Laskin Jeffrey D.

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference41 articles.

1. Paraquat: model for oxidant-initiated toxicity;Bus;Environ. Health Perspect.,1984

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3. Molecular mechanisms of pesticide-induced neurotoxicity: relevance to Parkinson's disease;Franco;Chem. Biol. Interact.,2010

4. Mechanisms of toxicity, clinical features, and management of diquat poisoning: a review;Jones;J. Toxicol. Clin. Toxicol.,2000

5. A literature review of paraquat toxicity;Onyeama;Vet. Hum. Toxicol.,1984

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