Mitochondrial free radical overproduction due to respiratory chain impairment in the brain of a mouse model of Rett syndrome: protective effect of CNF1-Reference-Cited by-同舟云学术

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Mitochondrial free radical overproduction due to respiratory chain impairment in the brain of a mouse model of Rett syndrome: protective effect of CNF1

Published:2015-06 Issue: Volume:83 Page:167-177
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

De Filippis Bianca,Valenti Daniela,de Bari Lidia,De Rasmo Domenico,Musto Mattia,Fabbri Alessia,Ricceri Laura,Fiorentini Carla,Laviola Giovanni,Vacca Rosa Anna^ORCID

Funder

Jerome Lejeune Foundation

AIRETT

Research (MIUR) Program FIRB-MERIT

IRSF HeART Award

Research (MIUR) Program FIRB Futuro in Ricerca

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference64 articles.

1. The story of Rett syndrome: from clinic to neurobiology;Chahrour;Neuron,2007

2. Clinical manifestations and stages of Rett syndrome;Hagberg;Ment. Retard. Dev. Disabil. Res. Rev.,2002

3. Social impairments in Rett syndrome: characteristics and relationship with clinical severity;Kaufmann;J. Intellect. Disabil. Res.,2012

4. Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2;Amir;Nat. Genet.,1999

5. A mouse Mecp2-null mutation causes neurological symptoms that mimic Rett syndrome;Guy;Nat. Genet.,2001

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