Overexpression of SLC25A51 promotes hepatocellular carcinoma progression by driving aerobic glycolysis through activation of SIRT5-Reference-Cited by-同舟云学术

Overexpression of SLC25A51 promotes hepatocellular carcinoma progression by driving aerobic glycolysis through activation of SIRT5

Published:2022-03 Issue: Volume:182 Page:11-22
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Bai Lu,Yang Zhao-Xu,Ma Peng-Fei,Liu Jian-Shan,Wang De-Sheng,Yu Heng-Chao^ORCID

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference39 articles.

1. Hepatocellular carcinoma;Forner;Lancet,2018

2. Epidemiology and risk factors of hepatocellular carcinoma;Budny;Pol. Merkur. Lek. : organ Polskiego Towarzystwa Lekarskiego,2017

3. Hepatocellular carcinoma: from diagnosis to treatment;Grandhi;Surg. Oncol.,2016

4. Management of hepatocellular carcinoma: prevention, surveillance, diagnosis, and staging;Ozer Etik;Exp. Clin. Transplant. Off. J. Middle East Soc. Organ Transplant.,2017

5. SLC25A51 is a mammalian mitochondrial NAD(+) transporter;Luongo;Nature,2020

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