Attenuation of oxidative damage by targeting mitochondrial complex I in neonatal hypoxic-ischemic brain injury-Reference-Cited by-同舟云学术

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Attenuation of oxidative damage by targeting mitochondrial complex I in neonatal hypoxic-ischemic brain injury

Published:2018-08 Issue: Volume:124 Page:517-524
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Kim Minso,Stepanova Anna,Niatsetskaya Zoya,Sosunov Sergey,Arndt Sabine,Murphy Michael P.,Galkin Alexander,Ten Vadim S.

Funder

NIH

MRC

Wellcome Trust Investigator award

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference38 articles.

1. Neonatal encephalopathy: an inadequate term for hypoxic-ischemic encephalopathy;Volpe;Ann. Neurol.,2012

2. Mitochondrial generation of reactive oxygen species after brain ischemia in the rat;Piantadosi;Stroke,1996

3. Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury;Starkov;Cell Calcium,2004

4. Hypoxic-ischemic injury in the developing brain: the role of reactive oxygen species originating in mitochondria;Ten;Neurol.Res.Int.,2012

5. Effect of reperfusion following cerebral ischaemia on the activity of the mitochondrial respiratory chain in the gerbil brain;Almeida;J. Neurochem.,1995

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