Low-dose oral copper treatment changes the hippocampal phosphoproteomic profile and perturbs mitochondrial function in a mouse model of Alzheimer's disease-Reference-Cited by-同舟云学术

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Low-dose oral copper treatment changes the hippocampal phosphoproteomic profile and perturbs mitochondrial function in a mouse model of Alzheimer's disease

Published:2019-05 Issue: Volume:135 Page:144-156
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Chen Chongyang,Jiang Xin,Li Yingchao,Yu Haitao,Li Shupeng,Zhang Zaijun,Xu Hua,Yang Ying,Liu Gongping,Zhu Feiqi,Ren Xiaohu,Zou Liangyu,Xu Benhong,Liu Jianjun,Spencer Peter S.,Yang Xifei

Funder

National Natural Science Foundation of China

Shenzhen Special Fund Project on Strategic Emerging Industry Development

Sanming Project of Medicine in Shenzhen

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference53 articles.

1. Astrocyte functions in the copper homeostasis of the brain;Scheiber;Neurochem. Int.,2013

2. Metabolism and functions of copper in brain;Scheiber;Prog. Neurobiol.,2014

3. Copper and iron disorders of the brain;Madsen;Annu. Rev. Neurosci.,2007

4. Role of copper in human neurological disorders;Desai;Am. J. Clin. Nutr.,2008

5. Role of copper in the onset of Alzheimer's disease compared to other metals;Bagheri;Front. Aging Neurosci.,2017

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1. Urolithin A improves motor dysfunction induced by copper exposure in SOD1 G93A transgenic mice via activation of mitophagy;2024-06-10

2. Phytochemicals targeting Alzheimer's disease via the AMP-activated protein kinase pathway, effects, and mechanisms of action;Biomedicine & Pharmacotherapy;2024-04

3. Insight into the effect of a heavy metal mixture on neurological damage in rats through combined serum metabolomic and brain proteomic analyses;Science of The Total Environment;2023-10

4. Quantitative Proteomic and Phosphoproteomic Analyses Reveal a Role of Death-Associated Protein Kinase 1 in Regulating Hippocampal Synapse;Molecular Neurobiology;2023-09-30

5. Low-dose Cu exposure enhanced α-synuclein accumulation associates with mitochondrial impairments in mice model of Parkinson's disease;Toxicology Letters;2023-09

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