m6A methyltransferase METTL3 contributes to sympathetic hyperactivity post-MI via promoting TRAF6-dependent mitochondrial ROS production-Reference-Cited by-同舟云学术

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m6A methyltransferase METTL3 contributes to sympathetic hyperactivity post-MI via promoting TRAF6-dependent mitochondrial ROS production

Published:2023-11 Issue: Volume:209 Page:342-354
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Yang Peijin,Wang Yu,Ge Weili,Jing Yanyan,Hu Hesheng,Yin Jie,Xue Mei,Wang Ye,Li Xiaolu,Li Xinran,Shi Yugen,Tan Jiayu,Li Yan,Yan Suhua

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference43 articles.

1. High-grade atrioventricular block in acute coronary syndromes: insights from the Global Registry of Acute Coronary Events;Singh;Eur. Heart J.,2015

2. Cardiac sympatho-vagal balance and ventricular arrhythmia;Kalla;Auton. Neurosci.,2016

3. Role of the sympathetic nervous system in the genesis of ventricular arrhythmia;Podrid;Circulation,1990

4. The relationship of autonomic imbalance, heart rate variability and cardiovascular disease risk factors;Thayer;Int. J. Cardiol.,2010

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1. Unravelling the impact of RNA methylation genetic and epigenetic machinery in the treatment of cardiomyopathy;Pharmacological Research;2024-09

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