Loss of pro-apoptotic Bax and Bak increases resistance to dihydroartemisinin-mediated cytotoxicity in normoxia but not in hypoxia in HCT116 colorectal cancer cells-Reference-Cited by-同舟云学术

Loss of pro-apoptotic Bax and Bak increases resistance to dihydroartemisinin-mediated cytotoxicity in normoxia but not in hypoxia in HCT116 colorectal cancer cells

Published:2021-10 Issue: Volume:174 Page:157-170
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Bader Sina,Wilmers Julia,Ontikatze Teona,Ritter Violetta,Jendrossek Verena,Rudner Justine^ORCID

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

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4. Activation of anti-oxidant Keap1/Nrf2 pathway modulates efficacy of dihydroartemisinin-based monotherapy and combinatory therapy with ionizing radiation;Bader;Free Radic. Biol. Med.,2021

5. Dihydroartemisinin is a hypoxia-active anti-cancer drug in colorectal carcinoma cells;Ontikatze;Front Oncol,2014

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