Dipeptide HCH6-1 inhibits neutrophil activation and protects against acute lung injury by blocking FPR1-Reference-Cited by-同舟云学术

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Dipeptide HCH6-1 inhibits neutrophil activation and protects against acute lung injury by blocking FPR1

Published:2017-05 Issue: Volume:106 Page:254-269
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Yang Shun-Chin,Chang Shih-Hsin,Hsieh Pei-Wen,Huang Yin-Ting,Ho Chiu-Ming,Tsai Yung-Fong,Hwang Tsong-Long

Funder

Yen Tjing Ling Medical Foundation

Ministry of Science Technology

Ministry of Education

Chang Gung Memorial Hospital

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference66 articles.

1. Neutrophils in the activation and regulation of innate and adaptive immunity;Mantovani;Nat. Rev. Immunol.,2011

2. Processes of sterile inflammation;Shen;J. Immunol.,2013

3. Neutrophils cascading their way to inflammation;Sadik;Trends Immunol.,2011

4. Neutrophil elastase inhibitors: a patent review and potential applications for inflammatory lung diseases (2010−2014);Tsai;Expert Opin. Ther. Pat.,2015

5. Blocking neutrophil integrin activation prevents ischemia-reperfusion injury;Yago;J. Exp. Med.,2015

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2. FPR1: A critical gatekeeper of the heart and brain;Pharmacological Research;2024-04

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4. FPR1 Antagonist (BOC-MLF) Inhibits Amniotic Epithelial-mesenchymal Transition;Current Medical Science;2024-02

5. Formylpeptide receptor 1 contributes to epidermal barrier dysfunction-induced skin inflammation through NOD-like receptor C4-dependent keratinocyte activation;British Journal of Dermatology;2023-11-18

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