Oxidative and ER stress-dependent ASK1 activation in steatotic hepatocytes and Kupffer cells sensitizes mice fatty liver to ischemia/reperfusion injury-Reference-Cited by-同舟云学术

Oxidative and ER stress-dependent ASK1 activation in steatotic hepatocytes and Kupffer cells sensitizes mice fatty liver to ischemia/reperfusion injury

Published:2017-11 Issue: Volume:112 Page:141-148
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Imarisio Chiara,Alchera Elisa,Bangalore Revanna Chandrashekar,Valente Guido,Follenzi Antonia,Trisolini Elena,Boldorini Renzo,Carini Rita

Funder

Fondazione Cariplo

Funds for Original Research of the Università del Piemonte Orientale

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference29 articles.

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3. Mechanisms of ischemic injury are different in the steatotic and normal rat liver;Selzner;Hepatology,2000

4. Mitochondrial uncoupling protein2 mediates steatotic liver injury following ischemia/reperfusion;Evans;J. Biol. Chem.,2008

5. Uncoupling protein2 (UCP2) induces mitochondrial proton leak and increases susceptibility of non-alcoholic steatohepatitis (NASH) liver to ischaemia-reperfusion injury;Serviddio;Gut,2008

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