Vasoactive intestinal peptide and peptide with N-terminal histidine and C-terminal isoleucine increase prolactin secretion in cultured rat pituitary cells (GE4C1) via a cAMP-dependent mechanism which involves transient elevation of intracellular Ca2+-Reference-Cited by-同舟云学术

Vasoactive intestinal peptide and peptide with N-terminal histidine and C-terminal isoleucine increase prolactin secretion in cultured rat pituitary cells (GE4C1) via a cAMP-dependent mechanism which involves transient elevation of intracellular Ca2+

Published:1987-02 Issue:2-3 Volume:49 Page:119-128
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Bjøro T.,Østberg B.C.,Sand O.,Gordeladze J.,Iversen J.-G.,Torjesen P.A.,Gautvik K.M.,Haug E.

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference49 articles.

1. Thyrotropin-releasing hormone-induced spike and plateau in cytosolic free Ca2+ concentrations in pituitary cells. Relation to prolactin release.

2. Porcine peptide having N-terminal histidine and C-terminal isoleucine amide (PHI)

3. The distributions of PHI and VIP in porcine gut and their co-localisation to a proportion of intrinsic ganglion cells

4. Vasoactive intestinal peptide causes a calcium-dependent stimulation of prolactin secretion in GH4C1 cells

5. Bjøro, T., Wiik, P., Opstad, P.C., Haug, E. and Gautvik, K.M. (submitted).

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