Multiple intracellular signallings are involved in thyrotropin-releasing hormone (TRH)-induced c-fos and jun B mRNA levels in clonal prolactin cells
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Molecular Biology,Biochemistry
Reference47 articles.
1. Possible role of Ca2+-independent protein kinase C isozyme, nPKCɛ, in thyrotropin- releasing hormone-stimulated signal transduction: Differential down-regulation of nPKCɛ in GH4C1 cells
2. Ionomycin acts as an ionophore to release TRH-regulated Ca2+ stores from GH4C1 cells
3. Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor
4. Calcium Induction of the mRNAs for Prolactin and c-fos Is Independent of Protein Kinase C Activity
5. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
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1. Regulation of Prolactin Gene Expression by Vasoactive Intestinal Peptide and Dopamine in the Turkey: Role of Ca2+ Signalling;Journal of Neuroendocrinology;2005-10
2. Structural and Functional Analysis of the Differential Effects of c-Jun and v-Jun on Prolactin Gene Expression;Molecular Endocrinology;2004-10-01
3. -Opioid Receptor Trafficking on Inhibitory Synapses in the Rat Brainstem;Journal of Neuroscience;2004-08-18
4. Thyrotropin-Releasing Hormone-Induced Down-Regulation of Pyroglutamyl Aminopeptidase II Activity Involvesl-Type Calcium Channels and Cam Kinase Activities in Cultures of Adenohypophyseal Cells;Journal of Neuroendocrinology;2002-03
5. MAP KINASE PHOSPHATASE-1 GENE EXPRESSION AND REGULATION IN NEUROENDOCRINE CELLS;Journal of Receptors and Signal Transduction;2002-01
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