p53 MUTATIONS IN PROSTATE CANCER BONE METASTASES SUGGEST THAT SELECTED p53 MUTANTS IN THE PRIMARY SITE DEFINE FOCI WITH METASTATIC POTENTIAL

Author:

NAVONE NORA M.1,LABATE MICHAEL E.1,TRONCOSO PATRICIA1,PISTERS LOUIS L.1,CONTI CLAUDIO J.1,VON ESCHENBACH ANDREW C.1,LOGOTHETIS CHRISTOPHER J.1

Affiliation:

1. Departments of Genitourinary Medical Oncology, Pathology and Urology, The University of Texas M. D. Anderson Cancer Center, Houston, Texas, and the Department of Carcinogenesis, Science Park-Research Division, The University of Texas M. D. Anderson Cancer Center, Smithville, Texas

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Urology

Reference31 articles.

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4. Analysis of genetic changes underlying local recurrence of prostate carcinoma during androgen deprivation therapy;Koivisto;Am. J. Pathol.,1995

5. Mutations in the androgen receptor gene are associated with progression of human prostate cancer to androgen independence;Tilley;Clin. Cancer Res.,1996

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